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Single-Session Distance Spikes Predict Running Injury Risk

27/10/2025

 
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For countless runners, the story is painfully familiar: you meticulously track your mileage, you listen to your body, and you follow the conventional wisdom, only to be sidelined by another frustrating injury. The most common advice centers on the "too much, too soon" theory, which suggests that overuse injuries happen when runners increase their training load too quickly. But what does "too much" or "too soon" actually mean? The ambiguity has left many runners guessing.

Now, a massive new study published in the British Journal of Sports Medicine involving over 5,200 runners has uncovered a surprising and counter-intuitive truth about what really causes overuse injuries. The findings challenge the very metrics many runners—and their GPS watches—rely on to stay safe, suggesting we've been focusing on the wrong thing all along.

Takeaway 1: The Real Danger Isn't Your Weekly Mileage—It's a Single Run.
The study's most significant finding is a major shift in how we should think about training load. It found that the biggest risk for an overuse injury doesn't come from a gradual increase in mileage over a week, but from a sudden spike in distance within a single running session.

The researchers propose a "single-session paradigm," identifying a specific risk threshold: running a single session that is more than 10% longer than your longest run in the preceding 30 days. Pushing past this 10% threshold was associated with a dramatic increase in injury rates.

The specific hazard rates are striking:
  • A 'small spike' (10% to 30% longer than your previous longest run) increased injury risk by 64%.
  • A 'moderate spike' (30% to 100% longer) increased risk by 52%.
  • A 'large spike' (more than 100% longer, or doubling the distance) increased risk by a staggering 128%.

While the risk for a 'moderate spike' appears slightly lower than for a 'small spike,' the overarching trend is clear: any jump in single-run distance greater than 10% significantly elevates injury risk, with the danger becoming most severe when doubling your distance.

This is a critical shift in thinking because musculoskeletal tissues like tendons and bones adapt gradually over weeks, but a single session that dramatically overloads them can cause micro-damage faster than the body can repair it, initiating an injury. It suggests that your one ambitious long run on the weekend could be far more dangerous than the total number of kilometers you accumulate over the week.

Takeaway 2: The Popular Training Metrics on Your Watch Might Be Misleading You.
Many dedicated runners rely on their wearable devices to manage training load using popular metrics like the week-to-week ratio or the Acute:Chronic Workload Ratio (ACWR). The study investigated these methods and found their effectiveness to be questionable, at best.

The results were surprising:
  • The week-to-week ratio (comparing one week's total mileage to the previous week's) showed no significant relationship with injury risk.
  • The Acute:Chronic Workload Ratio (ACWR) showed an inverse relationship—meaning that higher spikes in this ratio were actually associated with a decreased risk of injury.

This is deeply counter-intuitive and challenges the tools many runners use to prevent injury. The study's authors issued a strong word of caution in their summary of the findings:

Caution is advised when relying on recommended training load calculations such as the acute:chronic workload ratio and weekly- gradual changes, as no association, or even inverse associations, between these approaches and injury risk was found.

This finding is disruptive because it directly questions the scientific basis of features built into many popular GPS watches and training apps. This surprising result may suggest that runners who successfully handle large acute-to-chronic workload spikes are already highly resilient, or that the ACWR metric itself is poorly suited to capturing the specific, acute stress of a single long run that appears to be the real driver of injury.

Takeaway 3: A New, Simpler Rule to Guide Your Training.
Based on these powerful findings, the study's authors propose a new, evidence-based guideline for runners. It's a simple rule of thumb you can apply to any training plan:

Avoid running a distance in your current session that exceeds 10% of the longest distance you've covered in the previous 30 days.

For example, if the longest distance you've run in the last month is 10 kilometres, this new research suggests you should keep your next longest run under 11 kilometres to significantly reduce your injury risk.

It's important to note that even progressions under the 10% threshold aren't completely risk-free. The study found that jumps between 1% and 10% still correlated with a 19% higher injury rate, even if this figure wasn't statistically significant. Furthermore, this rule applies to a single session and doesn't account for the danger of stacking multiple progressions back-to-back without adequate recovery. Gradual, cautious progression is still the foundation of safe training.

Conclusion: Rethinking Your Next Long Run
For years, runners have been told to focus on gradual weekly increases in mileage. This landmark study suggests a paradigm shift is needed. The key to injury prevention may lie not in complex weekly load calculations, but in closely monitoring the acute stress of a single run. The real danger isn't just "too much, too soon," but "too long, right now."

It's important to note that these findings came from a study group that was predominantly male (nearly 78%), so while the principle is powerful, more research is needed to confirm these specific thresholds apply equally to all runners.

With this new insight, how will you plan your next big run differently?

​
​ARTICLE: 
  • How much running is too much? Identifying high-­ risk running sessions in a 5200-­ person cohort study

Ruptured Achilles Tendon: A Guide to Your Treatment Options

21/10/2025

 
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1. Understanding Your Injury and Your Choices

1.1. What is an Achilles Tendon Rupture?
An Achilles tendon rupture is a complete separation of the two ends of the tendon, which connects your calf muscle to your heel bone. This is one of the most common musculoskeletal injuries and often occurs during sports or other activities that involve a sudden change of direction, resulting in a forced dorsiflexion (upward bending) of the ankle. Patients frequently report hearing a "popping" sound and feeling as though they were kicked in the back of the heel at the moment of injury.

1.2. The Two Main Paths: Surgery vs. Non-Surgery
When you rupture your Achilles tendon, there are two primary treatment paths to consider, each with its own approach to healing the tendon:
  • Surgery: The two ends of the torn tendon are physically stitched back together during an operation in a hospital.
  • Non-surgical Management: The tendon is allowed to heal naturally, which involves avoiding an operation and beginning a structured rehabilitation program right away.

The best choice for any individual patient involves carefully balancing the different types of risks and benefits associated with each of these approaches.

​
2. The Core Trade-Off: Comparing the Key Risks
The central decision comes down to a clear trade-off: choosing the type of risk you are more comfortable with. While surgery lowers the risk of the tendon re-rupturing, it introduces risks related to the operation itself. Non-surgical treatment avoids operational risks but has a historically higher chance of re-rupture.

Primary Risks of Each Treatment Path

Non-Surgical Management:
Higher Risk of Re-Rupture
  • According to the data, 4 out of 100 people may re-rupture their tendon within 2 years.
  • The risk of re-rupture is the main drawback, though modern rehab protocols have significantly reduced this risk compared to older methods.
Lower Risk of Other Complications
  • According to the data, 2 out of 100 people may have other complications like poor ankle function.
  • This approach avoids the specific risks that come with an operation.

Surgical Management:
Lower Risk of Re-Rupture
  • According to the data, 2 out of 100 people may re-rupture their tendon within 2 years.
  • This is the primary advantage of surgery. The re-rupture rate is very low, cited at just 0.6% in one major study.
Higher Risk of Other Complications
  • According to the data, 5 out of 100 people may have other complications.
  • These complications are directly related to the surgery itself and most commonly include infection, wound healing problems, and sensory nerve injury (reported in 5.2% of patients for minimally invasive surgery in one study).

Given these different risk profiles, the next logical question is whether they lead to different functional outcomes for patients in the long run.

3. The End Goal: How Will You Recover?
While the initial treatment paths and their immediate risks differ, research shows that the final destination—long-term function and recovery—is remarkably similar for most patients.

3.1. The Surprising Finding: Similar Long-Term Function
Multiple high-quality studies, including large randomized trials and comprehensive meta-analyses, have reached the same conclusion: at the 12-month mark, there are no significant differences between the surgical and non-surgical groups across several key recovery metrics:
  • Patient-Reported Outcomes: Scores on functional questionnaires like the Achilles' tendon Total Rupture Score (ATRS) were similar for both groups.
  • Physical Performance: Objective tests measuring strength and jumping ability showed no material differences between treatments.
  • Ankle Motion: The range of motion in both dorsiflexion (flexing the foot up) and plantarflexion (pointing the foot down) was comparable.

3.2. Return to Work and Sports
The ability to get back to daily life is a critical measure of success. Here too, the outcomes are largely equivalent.
  • Return to Sports: Studies found no significant difference in patients' ability to return to sports at the same level they played before their injury, regardless of their treatment choice.
  • Return to Work: A nuanced finding from one moderate-quality study suggests that patients who undergo surgery may return to work approximately 7 weeks earlier than those treated non-surgically.

These findings suggest that for the average patient, both paths lead to a similar destination. The best route, therefore, often depends on specific individual factors.


4. Making the Decision: Key Factors to Discuss With Your Doctor
The choice between surgery and non-surgical care is not one-size-fits-all. It's a decision made in consultation with your doctor, weighing your unique anatomy, health, and the specifics of your injury.
  • The Tendon Gap: An ultrasound can be used to visualize the injury and measure the distance between the two ruptured tendon ends when the foot is pointed downwards (in equinus). Surgeons often recommend surgery if there is a 'large' distance or gap (e.g., greater than 5-10 mm), as this may affect the tendon's ability to heal properly on its own.
  • Your Health and Lifestyle: Patient-specific factors can significantly influence post-operative risks. Conditions like diabetes, smoking, or long-term steroid therapy are known to increase the risk of wound complications and infection after surgery. For these individuals, the higher risk of surgical complications may outweigh the benefits.
  • Accelerated Rehabilitation: The type of physical therapy you receive is critical. A meta-analysis revealed a crucial finding: when patients follow a modern "accelerated functional rehabilitation" protocol (which emphasizes early weight-bearing and movement), the difference in re-rupture rates between surgical and non-surgical treatment becomes statistically insignificant. This highlights that the quality of the rehabilitation plan is a powerful factor in determining success.

Ultimately, regardless of whether you start with surgery or a cast, the success of your recovery is heavily dependent on the one component that is the same for both paths: a dedicated rehabilitation program.

​
5. Your Path Forward: The Crucial Role of Rehabilitation
Rehabilitation is essential for a successful outcome, whether you have surgery or not. The process follows a very similar structure and timeline for both treatment groups, focusing on protecting the tendon as it heals and then gradually rebuilding strength and function.
  1. 0 to 3 months: The initial phase is focused on protecting the healing tendon. For the first 2 weeks, you will wear a cast or boot and cannot put weight on your foot. After that, weight-bearing is gradually introduced over the next 6 weeks, and you will start a gentle home-exercise program with a physiotherapist.
  2. 3 to 6-9 months: You will begin a more intense home-exercise program designed to further strengthen the leg and ankle, improve mobility, and restore function.
  3. By 9 months: Most patients can expect to make a gradual return to work and sports. This timeline is influenced by your individual health, the physical demands of your activities, and your personal recovery goals.

READ MORE:
  • Management of achilles tendon injury: A current concepts systematic review
  • Comparing Surgical and Conservative Treatment on Achilles Tendon Rupture: A Comprehensive Meta-Analysis of RCTs
  • Early controlled motion of the ankle compared with immobilisation in non-operative treatment of patients with an acute Achilles tendon rupture
  • Nonoperative or Surgical Treatment of Acute Achilles’ Tendon Rupture
  • Early Motion for Achilles Tendon Ruptures: Is Surgery Important?
  • I Ruptured My Achilles Tendon: Should I have Surgery?

Health News 20/10/25

20/10/2025

 
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  • More veg, less meat: the latest global update on a diet that’s good for people and the planet - 
  • Why do doctors run late? - 
  • Your body can be a portable gym: how to ditch membership fees and expensive equipment - 
  • Should I increase weights at the gym? How often? And by how much? - 
  • A sports dietitian’s simple rule for using electrolytes - 
  • Some US protein powders contain high levels of lead. Can I tell if mine is safe? - 
  • Sports Heat Tool helps sport manage heat risk - 
  • Should I take a magnesium supplement? Will it help me sleep or prevent muscle cramps? - 

Rethink Your Glute Training: 4 Surprising Truths From New Research

15/10/2025

 
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The fitness world is rightly obsessed with glute training. From improving athletic performance to building a powerful physique, the benefits of strong glutes are undeniable. Foundational exercises like squats, deadlifts, and the ever-popular barbell hip thrust have become the undisputed staples of countless leg-day routines. We perform them, we progress them, and we assume they represent the peak of glute development strategy.

But what if some of our most common assumptions are incomplete? As exercise science evolves, new research is providing surprising and often counter-intuitive insights that challenge this common wisdom. Studies that measure not just how a muscle feels, but how it activates, grows, and acutely impacts performance are painting a more nuanced picture of what truly works.

This article cuts through the noise to bring you four of the most impactful, science-backed takeaways from recent studies. Whether your goal is strength, size, or speed, these findings will help you train smarter, challenge your assumptions, and potentially redesign your entire approach to building better glutes.

1. The Surprising King of Glute Activation Isn't What You Think

In a fitness culture often focused on heavy barbells and complex machines, the barbell hip thrust has earned a legendary reputation for glute activation. However, against this backdrop of ever-heavier loads, a comprehensive systematic review published in the Journal of Sports Science and Medicine has identified a different, often-overlooked exercise as the top performer for firing up the glutes.

The exercise with the highest levels of demonstrated gluteus maximus (GMax) activation is the step-up. The 2020 review by Krause Neto et al. found that the step-up and its variations—including lateral, diagonal, and crossover step-ups—averaged an incredible 125% of Maximal Voluntary Isometric Contraction (MVIC, the gold-standard measure of a muscle's maximum electrical activity).

The likely reason for this superior activation is the multi-faceted demand of the movement. Step-ups are unilateral (single-leg) exercises that force the GMax to do more than just extend the hip. It must also work overtime to stabilize the pelvis and control the femur from adducting (moving inward) or rotating medially. This combined role of prime mover and stabilizer dramatically increases its total workload. This suggests that for pure neuromuscular stimulus, the load on the bar may be less important than the biomechanical demands of stabilizing the entire body on a single leg. For the lifter, this is your cue to treat step-ups not as a warm-up or accessory, but as a primary glute-strengthening movement worthy of progressive overload.

2. Adding Hip Thrusts Quantifiably Supercharges Glute Growth

While muscle activation studies are insightful, they only tell us about an exercise's potential. To know if an exercise truly builds muscle, we need long-term studies that directly measure hypertrophy (muscle growth). A 2024 study provides definitive proof of the barbell hip thrust's potent muscle-building effects.

In a study published in the International Journal of Strength and Conditioning, researchers Kassiano et al. compared two groups of untrained women over a 10-week training period. One group performed a routine consisting of 45º leg presses and stiff-leg deadlifts. The second group performed the exact same routine but added the barbell hip thrust.

The results were clear and quantifiable. The group that added the barbell hip thrust saw a +9.3% increase in gluteus maximus thickness. The group that performed the leg presses and deadlifts alone saw a +6.0% increase. But the key takeaway isn't just that "more is better." The insight lies in why it worked. The researchers noted that exercises like the leg press and stiff-leg deadlift impose peak forces when the glutes are at long muscle lengths (i.e., stretched). The hip thrust, in contrast, creates peak force when the glutes are at short muscle lengths (i.e., fully contracted). By adding the hip thrust, the second group wasn't just adding volume; they were adding a different stimulus that trained the glutes in a way the other exercises did not, leading to more complete development.

The current results suggest that performing barbell hip thrust, in addition to 45º leg press and stiff-leg deadlift, enhances muscle hypertrophy of the gluteus maximus compared to performing exclusively 45º leg press and stiff-leg deadlift.

3. Want to Be Faster? Do Hip Thrusts Right Before You Sprint

One of the most fascinating findings relates to an exercise's ability to boost immediate performance. This phenomenon, known as Post-Activation Performance Enhancement (PAPE), is an acute performance increase in an explosive movement (like a sprint) that occurs after performing a heavy conditioning activity.

Scientific research has confirmed that the hip thrust is a powerful tool for triggering PAPE. A systematic review by Krause Neto et al. (2019) noted that four separate studies found a significant improvement in sprint times immediately following a barbell hip thrust protocol. The mechanism is simple yet powerful: the heavy lift "wakes up" or primes the central nervous system, which allows for a more forceful and rapid muscle contraction in the subsequent sprint.

The key insight here is the direction of force. While a heavy squat also primes the nervous system, its force vector is primarily vertical—pushing up against gravity. Sprinting, however, is an expression of horizontal force—propelling your body forward. The hip thrust is unique in its ability to train this horizontal force vector, making it an exceptionally specific tool for improving acceleration. For athletes, the practical application is clear: sports science literature suggests that performing a few heavy hip thrusts during a warm-up, followed by a 5-8 minute rest period, could provide an immediate and measurable edge in speed. This rest period is critical; it allows the acute fatigue from the heavy lift to dissipate while keeping the nervous system in a heightened state of readiness.

4. The Hip Thrust's Overlooked Cousin is Just as Effective for Speed

While the hip thrust's effectiveness is well-established, new research highlights a simpler, more accessible alternative for achieving the same performance-enhancing benefits: the glute bridge.

A 2025 study by Çabuk et al. in the European Journal of Sport Science investigated the PAPE effects of both the hip thrust and the glute bridge on adolescent soccer players. Their research concluded that the glute bridge was also a highly effective tool for improving subsequent sprint performance.

These findings suggest that GB [glute bridge] exercises may offer a viable alternative to HT [hip thrust] exercises for eliciting PAPE effects, particularly in enhancing SP [sprint performance] and related mechanics in adolescent soccer players.

This is a significant practical finding. The glute bridge requires no bench and minimal setup, making it a convenient and powerful alternative for athletes in any setting, from a fully-equipped gym to a field-side warm-up. The study also revealed a subtle biomechanical difference: the hip thrust elicited greater activation in the vastus lateralis (a quadriceps muscle), whereas the glute bridge was associated with higher activation levels in the gluteus medius and gluteus maximus.

What does this mean for the athlete? An athlete needing to prime the entire hip and knee extension chain might prefer the hip thrust. However, an athlete wanting to focus the PAPE stimulus almost exclusively on the gluteal complex, or perhaps one recovering from quadriceps fatigue, might find the glute bridge to be a more precise and effective tool.

Conclusion: Rethink Your Next Leg Day

The science of strength is constantly advancing, providing us with better tools and clearer strategies. This isn't just a list of tips; it's a new framework for smarter glute training. Prioritize unilateral stability for activation (step-ups), drive growth with targeted, heavy hip extension that trains the muscle at both long and short lengths (hip thrusts plus other compounds), and strategically use these powerful movements to prime the nervous system for peak performance (PAPE). The research is clear: the humble step-up is the surprising king of activation, the hip thrust is a proven growth-booster, and both it and its cousin, the glute bridge, can make you measurably faster, instantly.

Now that you're armed with the latest science, how will you redesign your approach to building stronger, faster, and more powerful glutes?


​READ MORE: 
  • ​Gluteus Maximus Activation during Common Strength and Hypertrophy Exercises: A Systematic Review
  • The Acute Effects Of Hip Thrust and Glute Bridge Exercises With Different Loads on Sprint Performance and Horizontal Force–Velocity Profile
  • Barbell Hip Thrust, Muscular Activation and Performance: A Systematic Review
  • Addition of The Barbell Hip Thrust Elicits Greater Increases in Gluteus Maximus Muscle Thickness in Untrained Young Women​

Exercise Reduces Inflammation

14/10/2025

 
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The Exercise Paradox

Inflammation is a fundamental part of the body's defense system. When you get a cut, the familiar signs of redness, swelling, and heat are the work of your immune system aggressively fighting off infection and responding to tissue damage. This acute response is essential for healing. However, when inflammation fails to turn off, it can become a chronic, low-grade "silent menace" that contributes to a wide range of diseases, including diabetes, heart disease, and neurodegenerative conditions.

This brings us to a central paradox of health and wellness: exercise, a form of physical stress that causes temporary, acute inflammation in our muscles, is also one of our most powerful tools for combating the chronic, disease-causing type.

While many assume this benefit is simply a byproduct of weight loss, the science reveals a far more sophisticated and immediate relationship between movement and our immune system. This article explores five surprising, science-backed ways that exercise directly rewires your body to fight chronic inflammation.

The Anti-Inflammatory Effect Is Not Just About Weight Loss

It’s a common and logical assumption that exercise reduces inflammation primarily by reducing body fat. Adipose tissue, particularly visceral fat, is known to produce and release a range of inflammatory signals that contribute to a state of chronic, low-grade inflammation throughout the body.

However, research shows that exercise has powerful anti-inflammatory effects that are independent of any changes in body fat or Body Mass Index (BMI). For instance, a comprehensive review in the American Journal of Lifestyle Medicine concluded that exercise has potent, body fat–independent anti-inflammatory effects, making the benefits accessible regardless of weight loss. This is an incredibly empowering finding, as it makes the anti-inflammatory benefits of exercise accessible to everyone, no matter what the scale says. The positive changes are triggered by the act of moving, meaning every workout provides a direct anti-inflammatory benefit from day one.

Your Muscles Release Their Own Anti-Inflammatory "Myokines"

During exercise, your muscles do more than just contract; they act as endocrine organs, creating and releasing their own signaling molecules called "myokines" into the bloodstream. One of the most fascinating of these is Interleukin-6 (IL-6), which reveals a remarkable biological paradox.

In states of chronic disease, IL-6 is typically known as a pro-inflammatory cytokine. However, when IL-6 is released from contracting muscles during a workout, it behaves very differently. This fascinating paradox, detailed in reviews from researchers at institutions like Loughborough University, highlights a key mechanism of exercise's benefits. This exercise-induced IL-6 travels through the body and signals for the release of other, powerful anti-inflammatory cytokines, specifically IL-10 and IL-1 receptor antagonist (IL-1RA). In effect, the IL-6 produced by your muscles orchestrates a systemic, anti-inflammatory response that helps calm the immune system. This means your own muscles are capable of manufacturing and dispensing molecules that create a healing, inflammation-reducing environment throughout your body. This internal pharmacy isn't just for long-term health; its effects can be surprisingly immediate.

Just 20 Minutes Is Enough to Suppress Inflammation

The anti-inflammatory benefits of exercise are not just a long-term adaptation that occurs over months of training; they are an immediate physiological response. A study published in Brain, Behavior, and Immunity demonstrated that a single 20-minute session of moderate treadmill exercise had a significant, suppressive effect on the body's inflammatory response.

The mechanism was surprisingly direct. The workout caused a natural increase in catecholamines like epinephrine (adrenaline). This rise in epinephrine was shown to directly suppress the production of a key pro-inflammatory molecule called Tumor Necrosis Factor (TNF) in immune cells. This finding highlights that even short workouts are highly valuable for managing inflammation because they trigger an immediate chemical cascade that calms inflammatory pathways.

"Decreased inflammatory responses during acute exercise may protect against chronic conditions with low-grade inflammation."

Exercise Mobilizes Your Body's Inflammation-Fighting T-Cells

The physical exertion of a workout causes temporary, localized inflammation within the working muscles. Rather than letting this process run unchecked, the body has a built-in system to manage it and turn it into a source of strength and adaptation.

In response to this exertion-induced inflammation, the body mobilizes specialized immune cells called T-regulatory cells, or Tregs. A Harvard study conducted on mice found that these Tregs act as "peacekeepers," migrating directly to the muscle tissue to control the inflammation. With regular training, these cells not only subdue muscle damage but also help improve how muscles use energy, leading to enhanced endurance over time. This process shows how the body naturally leverages the stress of exercise to boost its own immune-regulating capacity, turning a potentially damaging event into a strengthening one.

"Our research suggests that with exercise, we have a natural way to boost the body’s immune responses to reduce inflammation." — Diane Mathis, Harvard Medical School

Running Fights Inflammation Inside Your Knees

Many people fear that running is inherently "bad for the knees" and will lead to joint degeneration. However, a pilot study on recreational runners suggests the opposite may be true. Researchers directly measured the biochemical environment inside the knee joint before and after a workout.

The findings directly countered the common assumption. A 30-minute run was shown to decrease the concentration of pro-inflammatory cytokines within the synovial fluid of the knee joint. Specifically, the levels of Granulocyte-macrophage colony-stimulating factor (GM-CSF) and Interleukin-15 (IL-15)—two molecules linked to the progression of joint disease—were lower after the run. Interestingly, the reduction in IL-15 was correlated with the number of foot strikes, suggesting that the mechanical loading itself is part of the beneficial signal. This research reframes running not as a destructive activity, but as one that may be chondroprotective by creating a less inflammatory environment inside the joint capsule.

A New Perspective on Movement

The anti-inflammatory power of exercise is far more sophisticated than just burning calories or reducing body fat. It is a multifaceted, immediate, and systemic process driven by elegant biological mechanisms. From muscles releasing their own anti-inflammatory medicine to the mobilization of specialized immune cells, exercise actively tunes our bodies for better health.

It is important to note, however, that more is not always better. This "elite athlete paradox" is a crucial reminder that the same systems we've discussed—like the release of immunomodulating cytokines—require balance. Extreme training without adequate recovery can push these anti-inflammatory signals too far, temporarily suppressing the immune system. For most of us, however, the message is clear: our bodies are designed to turn movement into a powerful anti-inflammatory signal.

Knowing that every step, jog, and stretch sends out these powerful signals, how might this change the way you think about your next workout?


READ MORE:
  • The Anti-Inflammatory Actions of Exercise Training
  • Inflammation and exercise: Inhibition of monocytic intracellular TNF production by acute exercise via b2-adrenergic activation
  • Running decreases knee intra‑articular cytokine and cartilage oligomeric matrix concentrations
  • Research shows working out gets inflammation-fighting T cells moving
  • The anti-inflammatory effects of exercise: mechanisms and implications for the prevention and treatment of disease

Health News 7/10/25

7/10/2025

 
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  • Do TikTok ‘anti-inflammatory diets’ really work? - 
  • CT scanners secretly waste more energy than used by a typical household – 
  • Not just ‘growing pains’: 1 in 5 Australian kids live with chronic pain, but it’s often invisible - 
  • Taller, leaner, faster: the evolution of the ‘perfect’ AFL body - 
  • Does ‘fasted’ cardio help you lose weight? Here’s the science - 
  • Experts unpack ‘quadrobics’, the fitness trend that claims leaping around on all fours will make you fit - 

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